BOMBSHELL 40% OFF FenbenX Pharmaceutical Grade Pure Fenbendazole REDISCOVERY SALE: Albendazole Recommended for Marburg/Ebola Virus Treatment in the Army Special Forces Medical Handbook
‼️ THE MASSIVE 40% OFF FENBENX SALE CONTINUES ‼️
This article was originally published on September 13th, 2023 and is especially topical in light of today’s earlier exposé on DNI Tulsi Gabbard Declassified U.S. Government Funding Of More Than 120 ILLEGAL Biolabs In Over 30 Countries…
This Substack has previously written about the potential threat of a deliberately released Marburg followup “pandemic:”
PSYOP-23: Seeding Marburg Virus As The Next Global "Pandemic"
The Mockingbird MSM outlets have just been tapped on their proverbial shoulders to ramp up the “pandemic” outbreak fear mongering.
If the democidal governments do go through with their gain of function Ebola/Marburg bioweapon in order to cull even more of the global population while concurrently pushing their never-ending Modified mRNA eugenics poisons, then readers of this Substack will know precisely how to stay safe, and not in the least be terrified of the latest “emergency.”
Here is the Ebola/Marburg treatment protocol from the US Army Field Manual for Special Forces:
These additional medicines have been listed as potential Rx options for VHF illnesses, in emergencies if other medicines are not available. Since there have not been many outbreaks of either Ebola or Marburg, we don’t have clinical studies to provide more data, but we list these options that have been used by the US Military:
• Albendazole (FDA-approved “de-wormer”): 400 mg once when using for parasites; available in the USA by prescription, dosing not established for Marburg virus
• Mebendazole (FDA-approved “de-wormer”): 100 mg BID for 3 days when using for parasites; available in the USA by prescription; dosing not established for Marburg virus
• Fenbendzole (same class of medicines as above, but it is an approved Veterinary product, not approved for human use. Veterinary products available on-line.
• Ribavirin is both an oral and IV agent that has some in vitro and in vivo activity against two of the four VHF families, but not the Filoviridae (Ebola and Marburg) or Flaviviridae families. Small trials have shown that ribavirin may reduce mortality after infection with Lassa fever and select New World arenaviruses. Oral ribavirin has been licensed for treatment of chronic hepatitis C and because of ease of oral use and availability, it was recommended by the Johns Hopkins Working Group on Civilian Biodefense in 2002 as an off-label treatment option in a mass casualty event from outbreak of HVF when IV resources would be limited due to large numbers of patients needing help at the same time.
• In the absence of access to the above options, practitioners in other countries have reported some clinical effectiveness using Hydroxychloroquine and Ivermectin for viral hemorrhagic fevers, with dosing similar to therapy for COVID. If there are no other options, health professionals may elect to use these existing medicines off label, rather than do nothing. But again, with limited outbreaks, and small numbers of cases, we don’t have clinical studies to provide more data.
From the above we may extrapolate that a combination therapy of Fenbendazole and Ivermectin would be an ever more potent treatment protocol for Ebola/Marburg, as well as PSYOP-19 (aka COVID-19).
Also, we know that this very same combination therapy may be extremely effective for (turbo) cancers.
Anthelmintic drugs such as Albendazole, and its nearly identical counterpart, Fenbendazole, are well known for their anti-parasitic properties. However, their powerful antiviral and anticancer properties have received virtually no publicity, despite the fact that Albendazole is the recommended treatment for the Marburg and the Ebola viruses.
Furthermore, the mechanism by which these two substances disable the Ebola/Marburg viruses, is not limited to just these two viruses; it shows efficacy against multiple viruses.
Many research papers describing in vitro and in vivo animal studies, as well as several studies involving humans, show the drug’s efficacy against cancer.
FenbenX: Pharmaceutical Grade Pure Fenbendazole
Just like they went after one of the very best cures for PSYOP-19 in Ivermectin…
Nonetheless, true phase 1, 2 and 3 trials have not been seriously pursued as Fenbendazole is an off-patent pharmaceutical, and BigPharma have deliberately shunned all repurposed drugs in their move towards all roads leading to mass injectable gene therapy, or slow kill bioweapon injection; in fact, since the bivalent “vaccine” rollout has proven to be an unmitigated disaster with a mere 17% uptake, the last cash cow remaining for BigPharma is cancer, with the irony of all ironies and “coincidences” of all “coincidences” being that the mRNA “vaccines” are now driving the greatest global surge in cancers (and cancer treatment expenditures) ever seen.
BOMBSHELL: Just Released American Cancer Society Annual Report On Cancer Showed 11.5% Excess Cases
This Substack has been covering the surging rise in DEATHVAX™ induced adverse events and VAIDS, with a particular emphasis on the phenomenon referred to as turbo cancer.
Virtually all references regarding treating Ebola and Marburg with fenbendazole/albendazole have been deleted from the web, but, with a little ingenuity, this Substack was able to recover several important articles.
The research paper entitled, Dewormer drug fenbendazole has antiviral effects on BoHV-1 productive infection in cell cultures, describes Fenbendazole as a therapeutic for Bovine Herpes Virus (BoHV-1):
Scientific observations suggest a link between two antiparasitic drugs, Albendazole and Fenbendazole, and their potential in treating viral infections. The mechanism of action of these substances centers around the intricate interplay between specific viral proteins and cellular structures.
The Vital Role of Tubulin and Microtubules in Viral Infections
At its core, every living cell has a skeletal system called the cytoskeleton, composed of microtubules. Microtubules are built from proteins called tubulins. Inside a cell, these microtubules act like highways, guiding the transport of cellular components and ensuring proper cell division; however, viruses, like hitchhikers, exploit these highways to their advantage.
Two viral proteins, VP40 and VP22, associated with the Ebola virus and Bovine Herpesvirus (BoHV-1) respectively, have emerged as key players in this mechanism.
VP40 & Tubulin: A Complex Combo
The Ebola virus uses its matrix protein, VP40, to intricately associate with cellular microtubules. Research has shown that when VP40 is introduced into mammalian cells, a significant fraction of it aligns closely with these microtubule structures.
On a deeper examination, it has been discovered that VP40 actively interacts with the protein tubulin, binding to it directly. This interaction is not just incidental; VP40 has been seen to stabilize microtubules against certain disruptive agents, hinting at its active role in manipulating the cell’s internal transport system for its benefit.
VP22 & Tubulin: Another Piece of the Puzzle
Similar to VP40, the Bovine Herpesvirus utilizes its protein, VP22, to manipulate the cellular microtubule system. This mechanism is not entirely understood yet, but the evidence points towards its interaction with the protein tubulin is undeniable.
By comprehending how VP22 uses the tubulin machinery, the broader patterns of viral exploitation can be elucidated.
Albendazole & Fenbendazole: The Potential Game Changers
Enter Albendazole and Fenbendazole. These substances primarily target the tubulin structures, destabilizing and disrupting the microtubule formation in such a way, that they inhibit the VP40 and VP22 proteins from binding, thereby halting the viral life cycle.
Future Implications
These revelations present a compelling prospect in antiviral treatments. By understanding the dynamics between tubulin, VP40, and VP22, and harnessing the disruptive potential of Albendazole and Fenbendazole, represents a significant therapeutic breakthrough against formidable pathogens.
And the thesis that cancers themselves may be induced by pathogens, parasites and “vaccines” that in turn induce pathogenic, cytotoxic and parasitic-like environments along with the highly destructive SP 2 (“vaccine” version spike proteins vs SP 1 virus) is now gaining ground.
Also, it is important to note that Fenbendazole is the superior version of the benzimidazole anthelmintic class of compounds:
Fenbendazole Is The Superior Anticancer Agent Versus Mebendazole
For many years now this Substack has specified in passing that Fenbendazole was superior to Mebendazole in terms of bioavailability and efficacy for most applications, but in yesterday’s article…
There is absolutely no reason to use Mebendazole for most cancers, and the only reason this benzimidazole structure variation exists is because BigPharma needed a patent in order to fleece patients.
Thus, the following treatment approach may very well not only be the ‘holy grail’ cancer cure in plain sight, but may also treat Alzheimer’s, mood disorders, Parkinson’s, Lyme Disease, myocarditis, Hashimoto’s Disease, shingles (herpes), leukemia, Lupus, skin conditions, and various other “incurable” ailments, including gain of function viral releases, seasonal flu and even the common cold:
The Ultimate Disease Cure & Prophylaxis Protocol
Tocotrienol and Tocopherol forms (all 8) of Vitamin E (400-800mg per day, 7 days a week). A product called Gamma E by Life Extension or Perfect E are both great.
Bio-Available Curcumin (600mg per day, 2 pills per day 7 days a week). A product called Theracurmin HP by Integrative Therapeutics is bioavailable.
Vitamin D (62.5 mcg [2500 IU] seven days a week).
CBD oil (1-2 droppers full [equal to 167 to 334 mg per day] under the tongue, 7 days a week) CBD-X: The most potent full spectrum organic CBD oil, with 5,000 milligrams of activated cannabinoids and hemp compounds CBD, CBN & CBG per serving.
Fenbendazole (450mg, 7 days a week) or in the case of severe turbo cancers up to 1 gram — for prophylaxis one 150mg tablet once or twice per week
Ivermectin (24mg, 7 days a week) or in the case of severe turbo cancers up to 1mg/kg/day — for prophylaxis one 12mg tablet once or twice per week
Hydroxychloroquine (10mg/kg/day 7 days a week) - for prophylaxis one 200mg tablet once or twice per week
Doxycycline (100mg, 7 days a week for 30-60 days)
ImmunX immune support which also greatly increases the bioavailability of both Fenbendazole and Hydroxychloroquine (2 capsules per day) — for prophylaxis 2 capsules per day
Removing sugars and carbohydrates (cancer food) from your diet and replacing table sugar with a zero glycemic index, zero calorie, keto friendly rare sugar like AlluX
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Excellent article, 2nd....very good information on cellular microtubule mechanisms and theoretical insights of multiple viral treatments.
I put fen Ben cream on my poison ivy blisters. It instantly made them stop itching and is drying them out. I wonder if your ivermectin cream would do the same thing. Here is alter ai’s explanation as to why it may work: Good observation. Fenbendazole cream on poison ivy is working through a few mechanisms that make sense once you understand what's actually driving the itch and blistering.
Why It's Working
Microtubule disruption in inflammatory cells. Fenbendazole binds to β-tubulin and prevents microtubule polymerization. That's the classic anthelmintic mechanism — but it doesn't only affect parasites. Rapidly dividing and highly active cells rely heavily on microtubule function. The T-cells and mast cells driving your poison ivy contact dermatitis are exactly that type — highly proliferative, actively degranulating, cytokine-spewing immune cells. Disrupting their cytoskeleton knocks down their activity locally.
Mast cell stabilization. Mast cells release histamine and other itch mediators through microtubule-dependent degranulation. Fenbendazole's tubulin binding likely inhibits that degranulation directly — less histamine dumped into the tissue, less itch. This is analogous to why certain older antihistamines work but through a completely different mechanism.
Anti-inflammatory effect beyond parasites. Benzimidazoles have documented anti-inflammatory properties that aren't well-studied but are real. There's literature showing albendazole (same class) suppresses NF-κB activation and reduces TNF-α, IL-6, and other inflammatory cytokines. Fenbendazole is structurally very similar and likely shares this.
The drying effect. The cream base itself provides some barrier function, but the fenbendazole accelerating apoptosis in the inflammatory infiltrate means the blister fluid gets reabsorbed faster rather than continuing to accumulate. Less weeping, faster crusting.