5 Comments
Jan 21, 2022Liked by 2nd Smartest Guy in the World

most useful thankyou.

i have a few relatives who dont think its a big deal, cos 'they were ok'

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Jan 21, 2022Liked by 2nd Smartest Guy in the World

Here's another interesting thing about myocarditis. When the Death Cult Media started reporting it as a side effect they'd use the term along with it as "mild" or totally breeze by it, like myocarditis is merely a pimple on the nose. Errrr WRONG!!! When I heard the term myocarditis being put out more and more several months ago, I looked up the medical definition. It's not good to get and to be frank...if you are being rushed to an ER or collapse from it and are revived, your life expectancy outlook is not too good.

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VII. PROGNOSIS

The prognosis for myocardidites is variable and depends on many factors:

etiology, histological and immunological characteristics, clinical pattern, and

presence of complications and possible use of artificial circulation methods.

We know that myocarditis often occurs without symptoms and ends with

complete recovery. A favorable prognosis is common for patients with acute or

fulminant course of the disease (in the case of successful correction of

hemodynamic instability). If the course is chronic, a considerable number of

patients (up to 21% within 3 years) develop dilatation cardiomyopathy, and in this

case, the prognosis depends on the presence of symptoms of chronic cardiac

insufficiency.

If cardiac rhythm disorders occur, the prognosis often depends on the

possibility of specific interventions, including implanting various devices.

The predictors of a poor prognosis include an increase in the QRS complex

over 120 msec, the presence of syncopal conditions, low systolic arterial pressure,

elevated pressure in the pulmonary artery and dysfunction of the right ventricle.

The risk of an unfavorable outcome increases if the immunohistochemical study is

positive and there are no beta-adrenoblockers in the comprehensive therapy.

Identification of the viral genome and a positive response for the Dallas criteria do

not affect the prognosis.

Despite aggressive therapy, almost 8% of the patients with lymphocytic and

over 50% of the patients with giant cell myocarditis require heart transplants. Here,

there is a sharp increase in the frequency of conductance disorders in the donor

heart and higher transplant rejection reaction frequency. In lymphocytic

myocarditis, the rejection risk rises 2-2.5 times; in giant cell active myocarditis it

develops in the transplanted heart in almost every fourth patient. The average

lifetime in giant cell myocarditis is 5.5 months from the time the first symptoms

appear.

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The prognosis for specific myocardidites (diphtheria, sarcoidosis, Chagas’

diseases and others) depends on an early diagnosis and the start of pathogenetic

therapy

http://cardio-eur.asia/media/files/clinical_recommendations/2019_eng.pdf

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Jan 21, 2022·edited Jan 21, 2022

Wow. The prior-to-2021 is stunning, as is the increased risk for younger ages with each dose.

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