I can vouch for that! In 2007 because I was volunteering at the Regional Hospital I was acquired to get the injection. Got the flu! Dispatched in 6 hours with 2 vitamin C, 2 325 mg Aspirin and 2 NAC. My bed sheets were soaking wet when I woke up. Last time I got the shot and I quit volunteering!
Jeez, the primary mechanism of action common to ALL of the viral mRNA products is this:
1. Common cell transfection reagents are used to introduce viral mRNA or the viral gene precursor of viral mRNA into healthy cells.
2. The cells automatically do three things:
A. The ribosomes of those cells translate the viral mRNA into many, MANY copies of a full length, biologically active viral protein that has no way of getting out of the product-compromised cells.
B. The cells have built-in internal sensors that can detect the presence of viral mRNA and viral protein inside the cells.
C. Once the cells have detected them, they send an alert to the innate immune system the products were designed to give the slip to. The collective alert is basically this: “We have detected the presence of viral mRNA and viral protein inside us. We have been compromised by viruses that have begun replication. Come and kill us before they can escape. It’s too late for us, but everybody else doesn’t have to die.”
In case you don’t know, this is EXACTLY what happens when you get infected with any virus capable of getting into any of your cells.
For respiratory viruses, it’s through a cell surface receptor on the outer-facing sides of cells of the respiratory epithelium.
The warning generated by the virally-compromised cells, starting with the very first one, is the first clue the innate immune system has about an infection being underway.
It gets into action as usual with proportional response; that is, it’s a limited, local, and small response that is driven directly by the signal strength.
If it’s sufficient, problem solved and you may never be aware of it.
But if the viral replication gets out of hand or you have no specific antibodies or T cells (previously made against any of the many epitopes on the viral coat proteins of that or related respiratory viruses) that can, at the very earliest stages post-breakout, inactivate the function of the viral coat proteins used to get into more cells or tag it for pickup and disposal (the antibodies) or to destroy it (the T cells), then the increased signal strength from more compromised cells recruits increasingly higher levels of innate immune inflammatory attacks.
The greater the level of attack, the greater the amount of inflammatory damage to nearby healthy cells. This is an unavoidable side effect of innate immune inflammatory attacks. This why the strategy, so to speak, of the innate immune system is defense in depth:
It responds with the least amount of force to end the threat in a way that inflicts the smallest amount of collateral damage.
Some viruses, though, have amino acid sequences in their coat proteins that will drive the innate immune system nuts enough to kill the host before the virus can so do by destroying enough of an organ to impair function past survivability. This happened with the 1918 flu from China. It happened with smallpox.
So, the goal of the innate immune system is to keep the response specific, targeted, and of an appropriate size to limit the spread of the virus while the innate immune system trains the adaptive immune system with bits and pieces of the viral proteins acquired from its action against the first virally-compromised cells.
What you feel when you’re coming down with a cold is not any effect of a virus but of a large enough innate immune response for you to notice.
Now, for really bad diseases that can severely damage or kill an normally healthy person (whether from effects the virus has or from the effects of your innate immune system responding, first, to the compromised cells’ alerts, and, second, to direct contact with viral proteins, whether or not attached to the virus), you can get exactly the same kind of adaptive immune response and by exactly the same mechanism as a viral infection, but without the innate immune damage, suffering, and dying part.
All you have to do is to present the innate system with a small load of viral proteins by injecting them into the extracellular fluid compartment of skeletal muscle.
When the innate immune system players encounter those antigens, they can tell they are viral or bacterial by characteristic amino acid sequences and then, without having to be triggered by any cell being compromised by a virus, just take it directly to the adaptive immune system to get it working on making specific antibodies and T cells.
The benefit?
Without having cells infected by viruses and calling up innate immune inflammatory attacks, you get the benefit of adaptive immune protection without any infection.
And then later, when you do get infected and your compromised cells start calling for innate immune inflammatory attacks after the first cells release the viruses into the extracellular fluid compartment, those viruses will be tagged and inactivated by antibodies and destroyed by T cells that are already in that fluid compartment.
And if a sufficient quantity and variety of viral antigens were used, you may never even know you had been infected or how many times it happens. Why? Because your innate immune system doesn’t have to be geared up enough for you to notice.
So that is what happens either in
A. the course of a viral infection in the absence of preexisting adaptive immune response or
B. the training of the adaptive immune system by the innate immune system using viral antigens acquired either by a battle against virally-compromised cells or by having the antigens presented to it from an external source.
Now observe what the viral mRNA companies do.
1. They used either a chimp cold virus (AstraZeneca) or a couple of other cell transfection reagents to infect healthy cells with a viral gene.
2. They did this in a way that bypassed notice by the innate immune system.
3. They did it in a way that did not keep the infection contained and local, like infection of only ACE2 receptor-expressing cells in the the respiratory tract.
4. AstraZeneca claimed its product was safer than the others because that replication-disabled chimp cold virus they used to transport the viral mRNA could enter only a specific population of cells having a unique receptor such as ACE2,
5. The other companies used indiscriminate cell transfection reagents that would enter any cell they bumped into in any organ,
6. “Yeah, but,” I can hear someone say. “They only shot it into your shoulder muscle.”
But AstraZeneca shot its chimp cold viruses into the shoulder muscle and they easily made their way from the extracellular fluid compartment that is continuously drained by the lymphatic system, that is dumped through a duct into the aorta near the heart, and that is then distributed throughout the body in seconds through the arterial system.
AstraZeneca’s chimp viruses found and infected a lot of other cells with that specific receptor that AstraZeneca claimed made their product safer than the others (just a positive spin way of saying “less dangerous”) that were in the heart and pericardial tissues. They caused so much innate immune inflammatory damage by the “Hey, we’ve been infected by a virus; come kill us” method described above that AstraZeneca was pulled off the market.
7. The other companies’ products are shot into the same shoulder muscle AstraZeneca’s chimp cold viruses were shot into.
They travel by diffusing and then through the lymphatic system to be dumped into the arterial system in exactly the same way and going to all the same places as AstraZeneca’s product, except AstraZeneca’s could get only into cells with a particular receptor used by the chimp virus, but the others can get into ANY cell.
8. Now your innate immune system will get the “We’ve been virally-compromised. Come kill us before it’s too late” and from many cells in multiple organ systems. That strong and broadly disseminated signal from all those different places will trigger very strong innate immune inflammatory attacks because it all looks like a sudden and massive viral infection appearing from out of the blue.
9. Those product-compromised cells have no way of exporting those spike proteins short of apoptosis or some other kind of spontaneous cell death or being attacked everywhere, all together, all at once by the innate immune system.
10. And then, once that happens multiple organ systems will be flooded with very large numbers of viral protein antigens that the innate immune system can now directly detect and react violently to, but now with a special “Yoo hoo, boys! Look at us over here” tag designed to kick up the innate immune system to even higher levels.
11. And it that weren’t bad enough, they are now up to at least the 7th so-called booster shot, and with each one you will have to go through exactly the same artificial infective trauma and the same over the top immunological responses but
A. With compounding levels of damage and
B. Without EVER developing a protective adaptive immune response.
12. “But,” I can hear the same folks say. “You’ll have an adaptive immune response. Like Pfizer said, ‘Our product only provides your body with the information it needs to build immunity,’ so see!”
Yeah, sure, a single set of antibodies against the epitopes on one lousy viral spike protein that maybe could protect you if you ever get infected by that particular variant, but not if you get infected by others. And certainly not if you get shot up with the viral mRNA products because they’re designed to NOT be noticed.
So when those products cause innate immune inflammatory attacks in the respiratory system, the symptoms you’ll have that are caused by the innate immune inflammatory attacks will look just like a cold or even pneumonia.
And this is why folks say that the viral mRNA products cause you to get infected repeatedly with Covid-19 and why flu shots are said to cause flu.
You are feeling the effects of your innate immune system responding to viral antigens, either in a genuine vaccine or to viral antigens the companies used their viral mRNA products to produce by using you as a liability-free bioreactor because they’re making you shoulder personally the manufacturing costs.
And when those innate immune inflammatory attacks happen in other organs? What then?
Why, then you’ll be told you are suffering from autoimmune attacks.
So, yeah, all of the top scientists in all those companies knew all about these things from long before they got their EUAs and the public has been left holding the stick.
And the reason I know this is because I’m a molecular biologist, have used these cell transfection reagents in the lab for years, understand the mechanism of action of viruses, vaccines, and viral mRNA products, and have over 30 years in the field of science.
These modified mRNA-LNP transfection injections are inherently dangerous, injurious, and deadly.
Every single doctor and scientist should have been able to recognize the immunological dangers of these mRNA shots.
I am still stunned and appalled that so many apparently did/do not because the immune system attack response, starting with the Killer T-cells targeting and destroying formerly healthy cells of tissues and organs inside the body that are now expressing foreign non-self proteins, was/is entirely predictable...and I am NOT a doctor nor am I in the medical field!
This was/is a PREDICTABLE IMMUNOLOGICAL CATASTROPHE.
As soon as I heard in 2020, “We’re going to use viral mRNA,” I knew that the primary mechanism of action would be that of viral infection.
I knew what the delivery vehicles they would use would have to be: certain methods originally developed for targeted delivery of pharmaceutical agents directly and only to the tissues in question (especially to tumors) that basically failed because they were not able to find anything that could target that well, but that someone else got the bright idea of using to deliver trans-genes into cells to express the proteins encoded by the genes for the purpose of studying the behavior of the protein in the context of a living cell.
And so appeared, scavenged from a failure, the extremely useful cell transfection reagents that, together with PCR, restriction enzymes, and DNA sequencing (in which I worked for almost 16 years before getting fired for refusing to further endanger an already innate immune-damaged heart), established the basis for modern molecular biology.
And of the 7 principal transfection reagents/techniques, guess which ones were used by these companies and why:
1. Electroporation. Using an electrical current to disrupt bacterial cell walls to make them leaky enough for replication vectors containing the target gene to drift into the cell.
2. Direct injection of the gene cargo into an individual cell using a micro needle.
3. The use of ultrasound to drive the gene cargo into cells.
4. The use of very strong magnetic fields to do what number 3 did.
5. The use of a replication-disabled adenovirus to introduce, by injection into skeletal muscle, the gene cargo into cells expressing the specific cell surface receptor that the adenovirus uses to gain entry for replication.
6. The use of inorganic nanoparticles precipated from a solution containing either pre-fabbed mRNA molecules or replication vectors with the target gene inserted to be introduced in the lab to immortal cell lines by dropping it into cell media or into people via injection into skeletal muscle.
7. The use of different moieties of lipid molecules containing the target gene replication vector to be introduced in the lab to immortal cell lines by dropping it into cell media or into people via injection into skeletal muscle.
Notice which of these required no equipment more specialized than a shot in the arm, could be done quickly on an outpatient basis, and appeared to look exactly like legitimate forms of vaccination people were already familiar with.
The last two were completely indiscriminate. When sticking to the outer surface of the cell membrane they were either endocytosed (the non-lipid nano particles) or they merged with the cell membrane’s lipid bilayer.
This made it really easy to do either transient or stable transfections.
Transient transfections using the nanoparticle method resulted in temporary but very strong expression of the desired protein.
Stable transfections using products like Lipo-Fect to introduce a replication vector containing the gene in question together with an antibiotic-resistance gene, allow you to weed out, by using antibiotics, all but those cells containing either the vector with the antibiotic resistance gene or the vector containing both that and the target gene.
As cells are passaged using growth media with antibiotic, all but those containing vectors with the antibiotic resistance are killed. As they continued to be passaged, eventually only those with replication vectors contained in the nucleus will survive.
The cells are split using a density that will ensure that colonies from single cells will grow adhered to the bottom of the flask.
When the colonies are big enough to be visible, they are picked using sterile swabs and put into separate flasks and grown in quantity.
Some may have had only a single vector in the nucleus some may have multiple copies. It’s possible by doing assays to see which is which. The ones most strongly producing the gene product without killing the cell are used because it’s easier to detect the effects of experiments.
The benefits of using immortal cell lines are: they are relatively easy to grow and they have no innate or adaptive immune systems to mess things up.
Doing it in vivo pretty much guarantees that cells of many different organs are going to be producing large quantities of proteins that are either physiologically useless or that are biologically active but being produced by cells completely outside the regulatory process that normally controls production.
For instance, if cells other than those in the pancreas were transfected with human insulin genes, the unregulated production of insulin would quickly lead to insulin coma and death.
And whereas cells in hair follicles producing keratin is okay, would you really want cells in the brain producing large amounts of keratin in an unregulated fashion?
And suppose you randomly introduced the gene for cholinesterase in cells not part of the regulatory apparatus of the neuromuscular junction.
Cholinesterase is released in a controlled fashion after a nerve impulse to get rid of the neurotransmitter acetylcholine.
If you did not have that, the neurotransmitter would build up, leading to convulsions and death.
Or if you had cholinesterase produced in very large quantities in an unregulated fashion, it would lead to nerve transition being blocked and paralysis and death.
And that would be from just using human genes for human proteins.
But when you use viral genes, then the innate immune system will necessarily and unavoidably be triggered to kill the cells producing it while harming many others in the process. And then the appearance of the viral protein outside the cells will lead to even more of the same.
The reason that legitimate viral protein vaccine boosters are given is because the adaptive immune response can be increased in a stepwise fashion without running the risk of too great an innate immune response to large quantities of viral antigen.
If you gave someone a large enough shot of viral protein antigens you could kill them by provoking an insanely high innate immune response.
The viral mRNA products can do exactly that.
And they were designed to deliberately cause innate immune attacks.
Why?
Because, except for infection with viral genes, the only other way the innate immune antigen-presenting cells will acquire viral protein antigens to train the adaptive immune system is by the use of a traditional viral protein vaccine, which those companies deliberately chose not to do.
Why?
Because
A. it’s really expensive and
B. there were existing very tough regulations controlling the production and use of such vaccines.
So they came up with something they claimed was so new and safe and special that the other regulations didn’t apply to it.
But it wasn’t new. It was the method used by viruses.
And it wasn’t safe. It provoked all the immune responses viruses did but in a more widespread and uncontrolled fashion.
And it wasn’t special. It was the opportunistic off-label use of cell transfection reagents.
ALL of the principal researchers at those companies knew ALL of these things. But they did it anyway.
And isn’t it more than a little weird that the companies, the lab coat wearing critics, the X-Files loony critics for years were all silent about the primary mechanism of action?
And isn’t it also weird that FB, that was hyperactively banning all sorts of Covid-related posts at the direction of federal agents from multiple agencies for years, in literally hundreds of posts I made in over ten different pages since the early part of 2021 about the primary mechanism common to ALL the viral mRNA products pulled one or two of my posts and then put them back up with an “Oops! We goofed!” message.
Why?
Because what I posted was all the most basic information about molecular biology, virology, and immunology and I used it all in the most Occam’s Razor fashion possible.
"... Because these viruses generally do not elicit complete and durable protective immunity by themselves, they have not to date been effectively controlled by licensed or experimental vaccines. ..."
-
Fast forward:
Effectiveness of the Influenza Vaccine During the 2024-2025 Respiratory Viral Season: A Prospective Cohort Study
"... there were already important past observations that suggested that respiratory infections at mucosal surfaces were susceptible to immune clearance by mechanisms not typical of infections caused by systemic (blood-borne) pathogens. Accordingly, it was likely to be important to understand the role for both innate and acquired immunity in response to viral infection, as well as the optimum acquired immune resistance mechanisms for viral clearance (B cell or antibody-mediated, versus T cell mediated)"
... and the publication prior to it related to Australia:
Analysis of SARS‐CoV‐2 haplotypes and genomic sequences during 2020 in Victoria, Australia, in the context of putative deficits in innate immune deaminase anti‐viral responses
At best the flu shots were useless, they were used to have the majority of the public practice lining up to have something injected into them. It also did the opposite, I attribute my initial resistance to the Covid shot propaganda to having to endure all the flu shot propaganda for decades.
I read an account by a woman who was deep into the Jim Jones cult. She recounted how they practiced drinking the kool-aid, just without the cyanide. They had regular drills.
I can vouch for that! In 2007 because I was volunteering at the Regional Hospital I was acquired to get the injection. Got the flu! Dispatched in 6 hours with 2 vitamin C, 2 325 mg Aspirin and 2 NAC. My bed sheets were soaking wet when I woke up. Last time I got the shot and I quit volunteering!
I’ve never taken nor will taken this killer vaccine. People, just eat good food, stay up on VitD3 with K2 and Magnesium. VitC , minerals.
Jeez, the primary mechanism of action common to ALL of the viral mRNA products is this:
1. Common cell transfection reagents are used to introduce viral mRNA or the viral gene precursor of viral mRNA into healthy cells.
2. The cells automatically do three things:
A. The ribosomes of those cells translate the viral mRNA into many, MANY copies of a full length, biologically active viral protein that has no way of getting out of the product-compromised cells.
B. The cells have built-in internal sensors that can detect the presence of viral mRNA and viral protein inside the cells.
C. Once the cells have detected them, they send an alert to the innate immune system the products were designed to give the slip to. The collective alert is basically this: “We have detected the presence of viral mRNA and viral protein inside us. We have been compromised by viruses that have begun replication. Come and kill us before they can escape. It’s too late for us, but everybody else doesn’t have to die.”
In case you don’t know, this is EXACTLY what happens when you get infected with any virus capable of getting into any of your cells.
For respiratory viruses, it’s through a cell surface receptor on the outer-facing sides of cells of the respiratory epithelium.
The warning generated by the virally-compromised cells, starting with the very first one, is the first clue the innate immune system has about an infection being underway.
It gets into action as usual with proportional response; that is, it’s a limited, local, and small response that is driven directly by the signal strength.
If it’s sufficient, problem solved and you may never be aware of it.
But if the viral replication gets out of hand or you have no specific antibodies or T cells (previously made against any of the many epitopes on the viral coat proteins of that or related respiratory viruses) that can, at the very earliest stages post-breakout, inactivate the function of the viral coat proteins used to get into more cells or tag it for pickup and disposal (the antibodies) or to destroy it (the T cells), then the increased signal strength from more compromised cells recruits increasingly higher levels of innate immune inflammatory attacks.
The greater the level of attack, the greater the amount of inflammatory damage to nearby healthy cells. This is an unavoidable side effect of innate immune inflammatory attacks. This why the strategy, so to speak, of the innate immune system is defense in depth:
It responds with the least amount of force to end the threat in a way that inflicts the smallest amount of collateral damage.
Some viruses, though, have amino acid sequences in their coat proteins that will drive the innate immune system nuts enough to kill the host before the virus can so do by destroying enough of an organ to impair function past survivability. This happened with the 1918 flu from China. It happened with smallpox.
So, the goal of the innate immune system is to keep the response specific, targeted, and of an appropriate size to limit the spread of the virus while the innate immune system trains the adaptive immune system with bits and pieces of the viral proteins acquired from its action against the first virally-compromised cells.
What you feel when you’re coming down with a cold is not any effect of a virus but of a large enough innate immune response for you to notice.
Now, for really bad diseases that can severely damage or kill an normally healthy person (whether from effects the virus has or from the effects of your innate immune system responding, first, to the compromised cells’ alerts, and, second, to direct contact with viral proteins, whether or not attached to the virus), you can get exactly the same kind of adaptive immune response and by exactly the same mechanism as a viral infection, but without the innate immune damage, suffering, and dying part.
All you have to do is to present the innate system with a small load of viral proteins by injecting them into the extracellular fluid compartment of skeletal muscle.
When the innate immune system players encounter those antigens, they can tell they are viral or bacterial by characteristic amino acid sequences and then, without having to be triggered by any cell being compromised by a virus, just take it directly to the adaptive immune system to get it working on making specific antibodies and T cells.
The benefit?
Without having cells infected by viruses and calling up innate immune inflammatory attacks, you get the benefit of adaptive immune protection without any infection.
And then later, when you do get infected and your compromised cells start calling for innate immune inflammatory attacks after the first cells release the viruses into the extracellular fluid compartment, those viruses will be tagged and inactivated by antibodies and destroyed by T cells that are already in that fluid compartment.
And if a sufficient quantity and variety of viral antigens were used, you may never even know you had been infected or how many times it happens. Why? Because your innate immune system doesn’t have to be geared up enough for you to notice.
So that is what happens either in
A. the course of a viral infection in the absence of preexisting adaptive immune response or
B. the training of the adaptive immune system by the innate immune system using viral antigens acquired either by a battle against virally-compromised cells or by having the antigens presented to it from an external source.
Now observe what the viral mRNA companies do.
1. They used either a chimp cold virus (AstraZeneca) or a couple of other cell transfection reagents to infect healthy cells with a viral gene.
2. They did this in a way that bypassed notice by the innate immune system.
3. They did it in a way that did not keep the infection contained and local, like infection of only ACE2 receptor-expressing cells in the the respiratory tract.
4. AstraZeneca claimed its product was safer than the others because that replication-disabled chimp cold virus they used to transport the viral mRNA could enter only a specific population of cells having a unique receptor such as ACE2,
5. The other companies used indiscriminate cell transfection reagents that would enter any cell they bumped into in any organ,
6. “Yeah, but,” I can hear someone say. “They only shot it into your shoulder muscle.”
But AstraZeneca shot its chimp cold viruses into the shoulder muscle and they easily made their way from the extracellular fluid compartment that is continuously drained by the lymphatic system, that is dumped through a duct into the aorta near the heart, and that is then distributed throughout the body in seconds through the arterial system.
AstraZeneca’s chimp viruses found and infected a lot of other cells with that specific receptor that AstraZeneca claimed made their product safer than the others (just a positive spin way of saying “less dangerous”) that were in the heart and pericardial tissues. They caused so much innate immune inflammatory damage by the “Hey, we’ve been infected by a virus; come kill us” method described above that AstraZeneca was pulled off the market.
7. The other companies’ products are shot into the same shoulder muscle AstraZeneca’s chimp cold viruses were shot into.
They travel by diffusing and then through the lymphatic system to be dumped into the arterial system in exactly the same way and going to all the same places as AstraZeneca’s product, except AstraZeneca’s could get only into cells with a particular receptor used by the chimp virus, but the others can get into ANY cell.
Continued:
8. Now your innate immune system will get the “We’ve been virally-compromised. Come kill us before it’s too late” and from many cells in multiple organ systems. That strong and broadly disseminated signal from all those different places will trigger very strong innate immune inflammatory attacks because it all looks like a sudden and massive viral infection appearing from out of the blue.
9. Those product-compromised cells have no way of exporting those spike proteins short of apoptosis or some other kind of spontaneous cell death or being attacked everywhere, all together, all at once by the innate immune system.
10. And then, once that happens multiple organ systems will be flooded with very large numbers of viral protein antigens that the innate immune system can now directly detect and react violently to, but now with a special “Yoo hoo, boys! Look at us over here” tag designed to kick up the innate immune system to even higher levels.
11. And it that weren’t bad enough, they are now up to at least the 7th so-called booster shot, and with each one you will have to go through exactly the same artificial infective trauma and the same over the top immunological responses but
A. With compounding levels of damage and
B. Without EVER developing a protective adaptive immune response.
12. “But,” I can hear the same folks say. “You’ll have an adaptive immune response. Like Pfizer said, ‘Our product only provides your body with the information it needs to build immunity,’ so see!”
Yeah, sure, a single set of antibodies against the epitopes on one lousy viral spike protein that maybe could protect you if you ever get infected by that particular variant, but not if you get infected by others. And certainly not if you get shot up with the viral mRNA products because they’re designed to NOT be noticed.
So when those products cause innate immune inflammatory attacks in the respiratory system, the symptoms you’ll have that are caused by the innate immune inflammatory attacks will look just like a cold or even pneumonia.
And this is why folks say that the viral mRNA products cause you to get infected repeatedly with Covid-19 and why flu shots are said to cause flu.
You are feeling the effects of your innate immune system responding to viral antigens, either in a genuine vaccine or to viral antigens the companies used their viral mRNA products to produce by using you as a liability-free bioreactor because they’re making you shoulder personally the manufacturing costs.
And when those innate immune inflammatory attacks happen in other organs? What then?
Why, then you’ll be told you are suffering from autoimmune attacks.
So, yeah, all of the top scientists in all those companies knew all about these things from long before they got their EUAs and the public has been left holding the stick.
And the reason I know this is because I’m a molecular biologist, have used these cell transfection reagents in the lab for years, understand the mechanism of action of viruses, vaccines, and viral mRNA products, and have over 30 years in the field of science.
Thank you!
These modified mRNA-LNP transfection injections are inherently dangerous, injurious, and deadly.
Every single doctor and scientist should have been able to recognize the immunological dangers of these mRNA shots.
I am still stunned and appalled that so many apparently did/do not because the immune system attack response, starting with the Killer T-cells targeting and destroying formerly healthy cells of tissues and organs inside the body that are now expressing foreign non-self proteins, was/is entirely predictable...and I am NOT a doctor nor am I in the medical field!
This was/is a PREDICTABLE IMMUNOLOGICAL CATASTROPHE.
As soon as I heard in 2020, “We’re going to use viral mRNA,” I knew that the primary mechanism of action would be that of viral infection.
I knew what the delivery vehicles they would use would have to be: certain methods originally developed for targeted delivery of pharmaceutical agents directly and only to the tissues in question (especially to tumors) that basically failed because they were not able to find anything that could target that well, but that someone else got the bright idea of using to deliver trans-genes into cells to express the proteins encoded by the genes for the purpose of studying the behavior of the protein in the context of a living cell.
And so appeared, scavenged from a failure, the extremely useful cell transfection reagents that, together with PCR, restriction enzymes, and DNA sequencing (in which I worked for almost 16 years before getting fired for refusing to further endanger an already innate immune-damaged heart), established the basis for modern molecular biology.
And of the 7 principal transfection reagents/techniques, guess which ones were used by these companies and why:
1. Electroporation. Using an electrical current to disrupt bacterial cell walls to make them leaky enough for replication vectors containing the target gene to drift into the cell.
2. Direct injection of the gene cargo into an individual cell using a micro needle.
3. The use of ultrasound to drive the gene cargo into cells.
4. The use of very strong magnetic fields to do what number 3 did.
5. The use of a replication-disabled adenovirus to introduce, by injection into skeletal muscle, the gene cargo into cells expressing the specific cell surface receptor that the adenovirus uses to gain entry for replication.
6. The use of inorganic nanoparticles precipated from a solution containing either pre-fabbed mRNA molecules or replication vectors with the target gene inserted to be introduced in the lab to immortal cell lines by dropping it into cell media or into people via injection into skeletal muscle.
7. The use of different moieties of lipid molecules containing the target gene replication vector to be introduced in the lab to immortal cell lines by dropping it into cell media or into people via injection into skeletal muscle.
Notice which of these required no equipment more specialized than a shot in the arm, could be done quickly on an outpatient basis, and appeared to look exactly like legitimate forms of vaccination people were already familiar with.
The last two were completely indiscriminate. When sticking to the outer surface of the cell membrane they were either endocytosed (the non-lipid nano particles) or they merged with the cell membrane’s lipid bilayer.
This made it really easy to do either transient or stable transfections.
Transient transfections using the nanoparticle method resulted in temporary but very strong expression of the desired protein.
Stable transfections using products like Lipo-Fect to introduce a replication vector containing the gene in question together with an antibiotic-resistance gene, allow you to weed out, by using antibiotics, all but those cells containing either the vector with the antibiotic resistance gene or the vector containing both that and the target gene.
As cells are passaged using growth media with antibiotic, all but those containing vectors with the antibiotic resistance are killed. As they continued to be passaged, eventually only those with replication vectors contained in the nucleus will survive.
The cells are split using a density that will ensure that colonies from single cells will grow adhered to the bottom of the flask.
When the colonies are big enough to be visible, they are picked using sterile swabs and put into separate flasks and grown in quantity.
Some may have had only a single vector in the nucleus some may have multiple copies. It’s possible by doing assays to see which is which. The ones most strongly producing the gene product without killing the cell are used because it’s easier to detect the effects of experiments.
The benefits of using immortal cell lines are: they are relatively easy to grow and they have no innate or adaptive immune systems to mess things up.
Doing it in vivo pretty much guarantees that cells of many different organs are going to be producing large quantities of proteins that are either physiologically useless or that are biologically active but being produced by cells completely outside the regulatory process that normally controls production.
For instance, if cells other than those in the pancreas were transfected with human insulin genes, the unregulated production of insulin would quickly lead to insulin coma and death.
And whereas cells in hair follicles producing keratin is okay, would you really want cells in the brain producing large amounts of keratin in an unregulated fashion?
And suppose you randomly introduced the gene for cholinesterase in cells not part of the regulatory apparatus of the neuromuscular junction.
Cholinesterase is released in a controlled fashion after a nerve impulse to get rid of the neurotransmitter acetylcholine.
If you did not have that, the neurotransmitter would build up, leading to convulsions and death.
Or if you had cholinesterase produced in very large quantities in an unregulated fashion, it would lead to nerve transition being blocked and paralysis and death.
And that would be from just using human genes for human proteins.
But when you use viral genes, then the innate immune system will necessarily and unavoidably be triggered to kill the cells producing it while harming many others in the process. And then the appearance of the viral protein outside the cells will lead to even more of the same.
The reason that legitimate viral protein vaccine boosters are given is because the adaptive immune response can be increased in a stepwise fashion without running the risk of too great an innate immune response to large quantities of viral antigen.
If you gave someone a large enough shot of viral protein antigens you could kill them by provoking an insanely high innate immune response.
The viral mRNA products can do exactly that.
And they were designed to deliberately cause innate immune attacks.
Why?
Because, except for infection with viral genes, the only other way the innate immune antigen-presenting cells will acquire viral protein antigens to train the adaptive immune system is by the use of a traditional viral protein vaccine, which those companies deliberately chose not to do.
Why?
Because
A. it’s really expensive and
B. there were existing very tough regulations controlling the production and use of such vaccines.
So they came up with something they claimed was so new and safe and special that the other regulations didn’t apply to it.
But it wasn’t new. It was the method used by viruses.
And it wasn’t safe. It provoked all the immune responses viruses did but in a more widespread and uncontrolled fashion.
And it wasn’t special. It was the opportunistic off-label use of cell transfection reagents.
ALL of the principal researchers at those companies knew ALL of these things. But they did it anyway.
And isn’t it more than a little weird that the companies, the lab coat wearing critics, the X-Files loony critics for years were all silent about the primary mechanism of action?
And isn’t it also weird that FB, that was hyperactively banning all sorts of Covid-related posts at the direction of federal agents from multiple agencies for years, in literally hundreds of posts I made in over ten different pages since the early part of 2021 about the primary mechanism common to ALL the viral mRNA products pulled one or two of my posts and then put them back up with an “Oops! We goofed!” message.
Why?
Because what I posted was all the most basic information about molecular biology, virology, and immunology and I used it all in the most Occam’s Razor fashion possible.
Let me introduce some evcidence.
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Why Do Vaccines Cause the Illnesses They Prevent
https://www.midwesterndoctor.com/p/why-do-vaccines-cause-the-illnesses
"Revealing The Forgotten Science of Vaccine Disease Provocation"
A Midwestern Doctor - Sep 05, 2025
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Rethinking next-generation vaccines for coronaviruses, influenzaviruses, and other respiratory viruses
https://www.cell.com/cell-host-microbe/fulltext/S1931-3128(22)00572-8
M. Morens; Jeffery K. Taubenberger; Anthony S. Fauci
CELL HOST & MICROBE – PERSPECTIVE - VOLUME 31, ISSUE 1, P146-157
DOI: https://doi.org/10.1016/j.chom.2022.11.016
JANUARY 11, 2023
"... Because these viruses generally do not elicit complete and durable protective immunity by themselves, they have not to date been effectively controlled by licensed or experimental vaccines. ..."
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Fast forward:
Effectiveness of the Influenza Vaccine During the 2024-2025 Respiratory Viral Season: A Prospective Cohort Study
https://www.medrxiv.org/content/10.1101/2025.01.30.25321421v4
Nabin K. Shrestha, Patrick C. Burke, Amy S. Nowacki, Steven M. Gordon
doi: https://doi.org/10.1101/2025.01.30.25321421
Oct. 11, 2025 – V4
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Spoiler: "Vaccines" for respiratory viruses simply don't work. ( and very likely do not work in no cases )
PROF. EDWARD J STEELE: "YOU CAN'T CHEAT THE UNIVERSE"
https://www.bitchute.com/video/AlPu8hAjMdUI/
18.10.2021
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The announced publication to it:
Nature of Acquired Immune Responses, Epitope Specificity and Resultant Protection from SARS-CoV-2
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8708741/
doi: 10.3390/jpm11121253
25.11.2021
Excerpt:
"... there were already important past observations that suggested that respiratory infections at mucosal surfaces were susceptible to immune clearance by mechanisms not typical of infections caused by systemic (blood-borne) pathogens. Accordingly, it was likely to be important to understand the role for both innate and acquired immunity in response to viral infection, as well as the optimum acquired immune resistance mechanisms for viral clearance (B cell or antibody-mediated, versus T cell mediated)"
... and the publication prior to it related to Australia:
Analysis of SARS‐CoV‐2 haplotypes and genomic sequences during 2020 in Victoria, Australia, in the context of putative deficits in innate immune deaminase anti‐viral responses
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8646704/
doi: 10.1111/sji.13100
30.09.2021
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Let`s get rid of this curse.
At best the flu shots were useless, they were used to have the majority of the public practice lining up to have something injected into them. It also did the opposite, I attribute my initial resistance to the Covid shot propaganda to having to endure all the flu shot propaganda for decades.
I read an account by a woman who was deep into the Jim Jones cult. She recounted how they practiced drinking the kool-aid, just without the cyanide. They had regular drills.